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    "textoCompleto" => "<span class="elsevierStyleSections"><p id="par0005" class="elsevierStylePara elsevierViewall">A 26-year-old Bolivian man residing in Spain for the past 12 years&#44; with no known history&#44; was admitted to the emergency department due to an episode compatible with a generalized self-limiting tonic&#8211;clonic seizure&#44; with no apparent trigger and no previous episodes&#46; Laboratory tests showed no ionic or red blood cell abnormalities&#44; just a C reactive protein &#40;CRP&#41; level of 7&#46;4<span class="elsevierStyleHsp" style=""></span>mg&#47;L&#46; An electroencephalogram showed no pathological abnormalities&#59; however&#44; computed tomography revealed cortical&#8211;subcortical cystic lesions in various locations &#40;<a class="elsevierStyleCrossRef" href="#fig0005">Fig&#46; 1</a>A&#41;&#46; Anticonvulsant treatment with levetiracetam was initiated&#44; and the patient was admitted for study&#46;</p><elsevierMultimedia ident="fig0005"></elsevierMultimedia><p id="par0010" class="elsevierStylePara elsevierViewall">We performed a serological study &#40;including <span class="elsevierStyleItalic">Strongyloides stercoralis</span>&#41;&#44; which was positive only for <span class="elsevierStyleItalic">Echinococcus granulosus&#59;</span> lymphocyte and immunoglobulin counts were normal&#46; Magnetic resonance imaging with contrast confirmed 10 cystic lesions&#44; predominantly in the subarachnoid spaces and cortical sulci&#44; with a hyperintense interior nodule and contrast enhancement &#40;<a class="elsevierStyleCrossRef" href="#fig0005">Fig&#46; 1</a>B and C&#41;&#44; compatible with neurocysticercosis in various evolutionary stages&#46; After an ophthalmic exam&#44; which ruled out ocular impairment&#44; and given the presence of active lesions&#44; we treated the patient with albendazole and praziquantel&#44; along with dexamethasone in a descending regimen and levetiracetam at minimum doses&#59; no new seizures were observed&#46;</p><p id="par0015" class="elsevierStylePara elsevierViewall">Neurocysticercosis is produced when an individual&#44; as an intermediate host&#44; is infected with the egg of the <span class="elsevierStyleItalic">Taenia solium</span> parasite through contaminated food or the fecal&#8211;oral pathway&#46; Neurocysticercosis is the most common cause of epileptic seizures in endemic areas and can present in up to 50&#37; of cases&#46; The diagnosis is performed according to radiological&#44; histological&#44; serological and epidemiological criteria&#46; Treatment is individualized depending on the number and activity of the lesions&#44; with albendazole and praziquantel the drugs of choice&#46;</p></span>"
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June - July 2020 Neurocysticercosis
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Vol. 220. Issue 5.
Pages 315-316 (June - July 2020)
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Vol. 220. Issue 5.
Pages 315-316 (June - July 2020)
Medicine in Images
DOI: 10.1016/j.rceng.2019.04.001
Neurocysticercosis
Neurocisticercosis
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A. Rosales-Castilloa,
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anrocas90@hotmail.com

Corresponding author.
, C. López-Roblesb, C. Fernández-Roldánb
a Servicio de Medicina Interna, Hospital Universitario Virgen de las Nieves, Granada, Spain
b Unidad de Enfermedades Infecciosas, Hospital Virgen de las Nieves, Granada, Spain
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